Across TCGA pan-cancer cohorts, C16orf87 Mutation is linked to patient survival in 3 of 34 cancer types, making it a survival-associated C16orf87 data layer compared with 24 for mass-spec protein and 1 for mass-spec protein.
The strongest signal is observed in liver hepatocellular carcinoma (LIHC), where higher C16orf87 Mutation is associated with worse disease-free survival. In most high-consensus cancer types, elevated C16orf87 expression acts as an unfavorable survival marker.
LIHC, STAD, and UCEC are the cancer types where C16orf87 Mutation most reproducibly stratifies survival.