BCL2 interacting protein 3 pseudogene 33Genealiases: []
Q-omics provides the consensus-scored BNIP3P33 profile across patient tissues and cancer cell-line models. BNIP3P33 expression is associated with patient survival in 15 of 34 cancer types, with the highest sampling consensus in ESCA. Among the 18 cancer types available for tumor–normal comparison, BNIP3P33 is differentially expressed in 5, with the highest sampling consensus in KIRC. Additionally, BNIP3P33 RNA expression shows 13,123 significant gene co-expression associations, with the highest sampling consensus in THYM. Together, these results highlight ESCA, KIRC, and THYM as cancer lineages where BNIP3P33 shows reproducible signals across survival, tumor–normal expression, and patient cross-omics analyses.
Every result is evaluated using two consensus scores. Sampling consensus measures how consistently a finding is reproduced within a cancer lineage across different conditions. Lineage consensus measures how broadly the result is shared across cancer types, distinguishing pan-cancer signals from lineage-specific patterns.
Premium analyses for BNIP3P33 — synthetic lethality, tumor antigen, and pembrolizumab response.
This table summarizes BNIP3P33 survival associations across molecular data types. BNIP3P33 RNA expression shows survival associations in the most cancer types (15). The rightmost column indicates the cancer type with the highest sampling consensus for each molecular layer.
This table ranks reproducible BNIP3P33 RNA expression–survival associations across cancer types. High BNIP3P33 expression shows unfavorable associations in LUAD, UCEC, LUSC and MESO, but favorable associations in ESCA and LIHC. The ESCA Kaplan–Meier curve shows clear separation, with the low-expression group declining faster, consistent with the favorable association (log-rank p = .002). Together, the overview and detailed table identify ESCA as the clearest survival context for BNIP3P33 RNA expression.
This table summarizes BNIP3P33 tumor–normal expression differences by data type. RNA shows broader differences across cancer types, with a lineage consensus of 5. The strongest signals are observed in KIRC for RNA.
This table ranks reproducible tumor–normal expression differences for BNIP3P33. A negative fold-change indicates higher expression in normal tissue than in tumor tissue. BNIP3P33 shows lower tumor expression in KIRC, KIRP, KICH, BRCA and PRAD. The KIRC box plot shows higher BNIP3P33 RNA expression in normal versus tumor tissue (log2 FC = −0.348, t-test p < 0.001).
This table shows molecular features associated with BNIP3P33 in patient tissues and cancer cell lines. In patient samples, BNIP3P33 shows the broadest associations at the RNA and protein expression levels, with THYM recurring as the lineage with the largest associated feature set.
