BMS1P4

Across TCGA pan-cancer cohorts, BMS1P4 RNA differs between tumor and matched normal tissue in 13 of 18 cancer types tested, making tumor–normal expression one of BMS1P4’s most consistent transcriptional readouts.

The strongest signal is observed in liver hepatocellular carcinoma (LIHC), where BMS1P4 RNA is more highly expressed in tumor relative to normal tissue. In most cancer types BMS1P4 is over-expressed in tumor, although a few such as KICH show the opposite, repressed pattern.

LIHC, KIRC, and COAD are the cancer types where BMS1P4 tumor–normal differential expression is most reproducible.

RNA tumor vs normal associations by lineage

Ranked by sampling consensus. Fold-change is the tumor-versus-normal difference in BMS1P4 RNA (log₂); positive values indicate higher expression in tumor. p-values are from the differential-expression test.

Lineage	Gender	Stage	Fold-change	p	Sampling consensus
LIHC	All	II,III,IV	+0.201	<.001	9	view →
KIRC	All	All	+0.114	<.001	9	view →
COAD	All	II,III,IV	+0.307	<.001	7	view →
UCEC	All	III,IV	+0.454	<.001	6	view →
BLCA	All	All	+0.331	.001	6	view →
HNSC	Male	All	+0.112	.009	5	view →
LUAD	All	All	+0.315	<.001	4	view →
STAD	All	All	+0.300	.008	4	view →
KICH	All	All	−0.252	.001	4	view →
CHOL	All	All	+0.670	<.001	3	view →
READ	Female	All	+0.383	.032	2	view →
PRAD	All	All	+0.212	<.001	2	view →

Pink = over-expressed in tumor, green = repressed in tumor. Showing the 12 strongest of 13 lineages.

BMS1P4–LIHC

Open the LIHC breakdown →