Q-omics provides the consensus-scored B3GALNT1P1 profile across patient tissues and cancer cell-line models. B3GALNT1P1 expression is associated with patient survival in 15 of 34 cancer types, with the highest sampling consensus in KIRC. Among the 18 cancer types available for tumor–normal comparison, B3GALNT1P1 is differentially expressed in 5, with the highest sampling consensus in THCA. Additionally, B3GALNT1P1 RNA expression shows 6,882 significant protein co-abundance associations, with the highest sampling consensus in HNSC. Together, these results highlight KIRC, THCA, and HNSC as cancer lineages where B3GALNT1P1 shows reproducible signals across survival, tumor–normal expression, and patient cross-omics analyses.
Every result is evaluated using two consensus scores. Sampling consensus measures how consistently a finding is reproduced within a cancer lineage across different conditions. Lineage consensus measures how broadly the result is shared across cancer types, distinguishing pan-cancer signals from lineage-specific patterns.
Premium analyses for B3GALNT1P1 — synthetic lethality, tumor antigen, and pembrolizumab response.
This table summarizes B3GALNT1P1 survival associations across molecular data types. B3GALNT1P1 RNA expression shows survival associations in the most cancer types (15). The rightmost column indicates the cancer type with the highest sampling consensus for each molecular layer.
This table ranks reproducible B3GALNT1P1 RNA expression–survival associations across cancer types. High B3GALNT1P1 expression shows unfavorable associations in KIRC, OV and THCA, but favorable associations in LUAD, HNSC and CESC. The KIRC Kaplan–Meier curve shows clear separation, with the high-expression group declining faster, consistent with the unfavorable association (log-rank p < 0.001). Together, the overview and detailed table identify KIRC as the clearest survival context for B3GALNT1P1 RNA expression.
This table summarizes B3GALNT1P1 tumor–normal expression differences by data type. RNA shows broader differences across cancer types, with a lineage consensus of 5. The strongest signals are observed in THCA for RNA.
This table ranks reproducible tumor–normal expression differences for B3GALNT1P1. A negative fold-change indicates higher expression in normal tissue than in tumor tissue. B3GALNT1P1 shows lower tumor expression in THCA, LUAD, KIRP, KIRC and KICH. The THCA box plot shows higher B3GALNT1P1 RNA expression in normal versus tumor tissue (log2 FC = −0.106, t-test p < 0.001).
This table shows molecular features associated with B3GALNT1P1 in patient tissues and cancer cell lines. In patient samples, B3GALNT1P1 shows the broadest associations at the RNA and protein expression levels, with HNSC recurring as the lineage with the largest associated feature set.