ATP13A5-AS1

Across TCGA pan-cancer cohorts, ATP13A5-AS1 RNA differs between tumor and matched normal tissue in 6 of 18 cancer types tested, making tumor–normal expression one of ATP13A5-AS1’s most consistent transcriptional readouts.

The strongest signal is observed in lung adenocarcinoma (LUAD), where ATP13A5-AS1 RNA is more highly expressed in tumor relative to normal tissue. In most cancer types ATP13A5-AS1 is over-expressed in tumor, although a few such as HNSC and THCA show the opposite, repressed pattern.

LUAD, KIRC, and LUSC are the cancer types where ATP13A5-AS1 tumor–normal differential expression is most reproducible.

RNA tumor vs normal associations by lineage

Ranked by sampling consensus. Fold-change is the tumor-versus-normal difference in ATP13A5-AS1 RNA (log₂); positive values indicate higher expression in tumor. p-values are from the differential-expression test.

Lineage	Gender	Stage	Fold-change	p	Sampling consensus
LUAD	All	All	+0.167	<.001	5	view →
KIRC	All	All	+0.076	.004	5	view →
LUSC	All	All	+0.300	<.001	3	view →
UCEC	All	All	+0.313	.011	2	view →
HNSC	Male	All	−0.268	.024	2	view →
THCA	All	All	−0.111	.010	2	view →

Pink = over-expressed in tumor, green = repressed in tumor. Showing the 6 strongest of 6 lineages.

ATP13A5-AS1–LUAD

Open the LUAD breakdown →