Q-omics provides the consensus-scored ASNSP1 profile across patient tissues and cancer cell-line models. ASNSP1 expression is associated with patient survival in 23 of 34 cancer types, with the highest sampling consensus in COAD. Among the 18 cancer types available for tumor–normal comparison, ASNSP1 is differentially expressed in 8, with the highest sampling consensus in LIHC. Additionally, ASNSP1 RNA expression shows 9,895 significant gene co-expression associations, with the highest sampling consensus in THYM. Together, these results highlight COAD, LIHC, and THYM as cancer lineages where ASNSP1 shows reproducible signals across survival, tumor–normal expression, and patient cross-omics analyses.
Every result is evaluated using two consensus scores. Sampling consensus measures how consistently a finding is reproduced within a cancer lineage across different conditions. Lineage consensus measures how broadly the result is shared across cancer types, distinguishing pan-cancer signals from lineage-specific patterns.
Premium analyses for ASNSP1 — synthetic lethality, tumor antigen, and pembrolizumab response.
This table summarizes ASNSP1 survival associations across molecular data types. ASNSP1 RNA expression shows survival associations in the most cancer types (23). The rightmost column indicates the cancer type with the highest sampling consensus for each molecular layer.
This table ranks reproducible ASNSP1 RNA expression–survival associations across cancer types. High ASNSP1 expression shows unfavorable associations in COAD, KIRC, UVM, LIHC, KIRP and STAD. The COAD Kaplan–Meier curve shows clear separation, with the high-expression group declining faster, consistent with the unfavorable association (log-rank p < 0.001). Together, the overview and detailed table identify COAD as the clearest survival context for ASNSP1 RNA expression.
This table summarizes ASNSP1 tumor–normal expression differences by data type. RNA shows broader differences across cancer types, with a lineage consensus of 8. The strongest signals are observed in LIHC for RNA.
This table ranks reproducible tumor–normal expression differences for ASNSP1. A negative fold-change indicates higher expression in normal tissue than in tumor tissue. ASNSP1 shows lower tumor expression in PAAD and higher tumor expression in LIHC, LUSC, BRCA, BLCA and LUAD. The LIHC box plot shows higher ASNSP1 RNA expression in tumor versus normal tissue (log2 FC = +1.132, t-test p < 0.001).
This table shows molecular features associated with ASNSP1 in patient tissues and cancer cell lines. In patient samples, ASNSP1 shows the broadest associations at the RNA and protein expression levels, with THYM recurring as the lineage with the largest associated feature set. In cancer cell lines, ASNSP1 RNA and mutation anchors are most strongly linked to RNA-expression features, especially in LUNG_NSCLC_LUAD, while CRISPR and shRNA rows add functional-dependency signals in SKIN.
