ASB16-AS1

Across TCGA pan-cancer cohorts, ASB16-AS1 RNA differs between tumor and matched normal tissue in 13 of 18 cancer types tested, making tumor–normal expression one of ASB16-AS1’s most consistent transcriptional readouts.

The strongest signal is observed in kidney renal papillary cell carcinoma (KIRP), where ASB16-AS1 RNA is more highly expressed in tumor relative to normal tissue. In most cancer types ASB16-AS1 is over-expressed in tumor, although a few such as KICH show the opposite, repressed pattern.

KIRP, LIHC, and HNSC are the cancer types where ASB16-AS1 tumor–normal differential expression is most reproducible.

RNA tumor vs normal associations by lineage

Ranked by sampling consensus. Fold-change is the tumor-versus-normal difference in ASB16-AS1 RNA (log₂); positive values indicate higher expression in tumor. p-values are from the differential-expression test.

Lineage	Gender	Stage	Fold-change	p	Sampling consensus
KIRP	All	II,III,IV	+0.703	<.001	11	view →
LIHC	Female	II,III,IV	+1.502	<.001	9	view →
HNSC	Male	III,IV	+0.688	<.001	9	view →
LUAD	Female	All	+0.511	<.001	7	view →
COAD	All	II,III,IV	+0.459	<.001	6	view →
BRCA	All	II,III,IV	+0.394	<.001	6	view →
CHOL	Male	All	+2.116	<.001	5	view →
BLCA	Female	III,IV	+0.824	.026	5	view →
LUSC	All	All	+0.498	<.001	4	view →
KIRC	All	All	+0.192	.003	4	view →
KICH	All	All	−0.393	.001	3	view →
ESCA	All	All	+0.721	.021	2	view →

Pink = over-expressed in tumor, green = repressed in tumor. Showing the 12 strongest of 13 lineages.

ASB16-AS1–KIRP

Open the KIRP breakdown →