Across TCGA pan-cancer cohorts, ARHGAP42P1 RNA differs between tumor and matched normal tissue in 4 of 18 cancer types tested, making tumor–normal expression one of ARHGAP42P1’s most consistent transcriptional readouts.

The strongest signal is observed in pancreatic adenocarcinoma (PAAD), where ARHGAP42P1 RNA is repressed in tumor relative to normal tissue. In most cancer types ARHGAP42P1 is over-expressed in tumor, although a few such as PAAD and STAD show the opposite, repressed pattern.

PAAD, PRAD, and LUSC are the cancer types where ARHGAP42P1 tumor–normal differential expression is most reproducible.

RNA tumor vs normal associations by lineage

Ranked by sampling consensus. Fold-change is the tumor-versus-normal difference in ARHGAP42P1 RNA (log₂); positive values indicate higher expression in tumor. p-values are from the differential-expression test.

Lineage	Gender	Stage	Fold-change	p	Sampling consensus
PAAD	Female	All	−0.050	.037	2	view →
PRAD	All	All	+0.050	.001	2	view →
LUSC	Male	All	+0.024	.015	2	view →
STAD	Female	III,IV	−0.050	.037	1	view →

Pink = over-expressed in tumor, green = repressed in tumor. Showing the 4 strongest of 4 lineages.

Exploration

Same gene, other analysis: ARHGAP42P1 RNA expression & survival · all ARHGAP42P1 data types