Across TCGA pan-cancer cohorts, ARHGAP16P RNA differs between tumor and matched normal tissue in 2 of 18 cancer types tested, making tumor–normal expression one of ARHGAP16P’s most consistent transcriptional readouts.
The strongest signal is observed in breast invasive carcinoma (BRCA), where ARHGAP16P RNA is more highly expressed in tumor relative to normal tissue. In most cancer types ARHGAP16P is over-expressed in tumor.
BRCA and LUAD are the cancer types where ARHGAP16P tumor–normal differential expression is most reproducible.