Q-omics provides the consensus-scored ALDOAP1 profile across patient tissues and cancer cell-line models. ALDOAP1 expression is associated with patient survival in 20 of 34 cancer types, with the highest sampling consensus in BRCA. Among the 18 cancer types available for tumor–normal comparison, ALDOAP1 is differentially expressed in 8, with the highest sampling consensus in KIRC. Additionally, ALDOAP1 RNA expression shows 8,833 significant gene co-expression associations, with the highest sampling consensus in READ. Together, these results highlight BRCA, KIRC, and READ as cancer lineages where ALDOAP1 shows reproducible signals across survival, tumor–normal expression, and patient cross-omics analyses.
Every result is evaluated using two consensus scores. Sampling consensus measures how consistently a finding is reproduced within a cancer lineage across different conditions. Lineage consensus measures how broadly the result is shared across cancer types, distinguishing pan-cancer signals from lineage-specific patterns.
Premium analyses for ALDOAP1 — synthetic lethality, tumor antigen, and pembrolizumab response.
This table summarizes ALDOAP1 survival associations across molecular data types. ALDOAP1 RNA expression shows survival associations in the most cancer types (20). The rightmost column indicates the cancer type with the highest sampling consensus for each molecular layer.
This table ranks reproducible ALDOAP1 RNA expression–survival associations across cancer types. High ALDOAP1 expression shows unfavorable associations in BRCA and PAAD, but favorable associations in SKCM, KIRC, HNSC and COAD. The BRCA Kaplan–Meier curve shows clear separation, with the high-expression group declining faster, consistent with the unfavorable association (log-rank p < 0.001). Together, the overview and detailed table identify BRCA as the clearest survival context for ALDOAP1 RNA expression.
This table summarizes ALDOAP1 tumor–normal expression differences by data type. RNA shows broader differences across cancer types, with a lineage consensus of 8. The strongest signals are observed in KIRC for RNA.
This table ranks reproducible tumor–normal expression differences for ALDOAP1. A negative fold-change indicates higher expression in normal tissue than in tumor tissue. ALDOAP1 shows lower tumor expression in THCA and higher tumor expression in KIRC, COAD, BRCA, LUAD and LUSC. The KIRC box plot shows higher ALDOAP1 RNA expression in tumor versus normal tissue (log2 FC = +0.175, t-test p < 0.001).
This table shows molecular features associated with ALDOAP1 in patient tissues and cancer cell lines. In patient samples, ALDOAP1 shows the broadest associations at the RNA and protein expression levels, with READ recurring as the lineage with the largest associated feature set.
