Across TCGA pan-cancer cohorts, ACTG1P12 RNA differs between tumor and matched normal tissue in 3 of 18 cancer types tested, making tumor–normal expression one of ACTG1P12’s most consistent transcriptional readouts.

The strongest signal is observed in colon adenocarcinoma (COAD), where ACTG1P12 RNA is more highly expressed in tumor relative to normal tissue. In most cancer types ACTG1P12 is over-expressed in tumor, although a few such as KICH show the opposite, repressed pattern.

COAD, LUAD, and KICH are the cancer types where ACTG1P12 tumor–normal differential expression is most reproducible.

RNA tumor vs normal associations by lineage

Ranked by sampling consensus. Fold-change is the tumor-versus-normal difference in ACTG1P12 RNA (log₂); positive values indicate higher expression in tumor. p-values are from the differential-expression test.

Lineage	Gender	Stage	Fold-change	p	Sampling consensus
COAD	Female	All	+0.684	<.001	5	view →
LUAD	All	All	+0.108	.003	5	view →
KICH	Female	II,III,IV	−0.152	.002	3	view →

Pink = over-expressed in tumor, green = repressed in tumor. Showing the 3 strongest of 3 lineages.

Exploration

Same gene, other analysis: ACTG1P12 RNA expression & survival · all ACTG1P12 data types