ACTBP13

associated omics data
Gene

Q-omics provides the consensus-scored ACTBP13 profile across patient tissues and cancer cell-line models. ACTBP13 expression is associated with patient survival in 19 of 34 cancer types, with the highest sampling consensus in BLCA. Among the 18 cancer types available for tumor–normal comparison, ACTBP13 is differentially expressed in 8, with the highest sampling consensus in BRCA. Additionally, ACTBP13 RNA expression shows 9,174 significant gene co-expression associations, with the highest sampling consensus in ESCA. Together, these results highlight BLCA, BRCA, and ESCA as cancer lineages where ACTBP13 shows reproducible signals across survival, tumor–normal expression, and patient cross-omics analyses.

Every result is evaluated using two consensus scores. Sampling consensus measures how consistently a finding is reproduced within a cancer lineage across different conditions. Lineage consensus measures how broadly the result is shared across cancer types, distinguishing pan-cancer signals from lineage-specific patterns.

Survival associations

This table summarizes ACTBP13 survival associations across molecular data types. ACTBP13 RNA expression shows survival associations in the most cancer types (19). The rightmost column indicates the cancer type with the highest sampling consensus for each molecular layer.
ACTBP13 data typeSurvival analysisLineage consensusLineage of highest sampling consensus
RNAKaplan–Meier19BLCA (81)view →
This table ranks reproducible ACTBP13 RNA expression–survival associations across cancer types. High ACTBP13 expression shows unfavorable associations in LIHC, KIRC, THYM and LUAD, but favorable associations in BLCA and STAD. The BLCA Kaplan–Meier curve shows clear separation, with the low-expression group declining faster, consistent with the favorable association (log-rank p = .001). Together, the overview and detailed table identify BLCA as the clearest survival context for ACTBP13 RNA expression.
LineageMeasureSplitStageAUC1
high
AUC2
low
pSampling consensus
BLCAOSMedianAll0.5220.243.00181view →
LIHCDFSTertileAll0.2750.493<.00148view →
KIRCDFSQuartileAll0.6020.837<.00140view →
THYMDFSMedianII,III,IV0.5730.903.00632view →
STADDFSQuartileII,III,IV0.7740.448.00428view →
LUADOSQuartileII,III,IV0.7000.893.01415view →
Pink = unfavorable, green = favorable. all 19 lineages →

ACTBP13-BLCA (OS)

Kaplan–Meier survival curve for ACTBP13 RNA expression in BLCA: high vs low expression groups.

Explore this curve interactively →

Tumor vs Normal expression

This table summarizes ACTBP13 tumor–normal expression differences by data type. RNA shows broader differences across cancer types, with a lineage consensus of 8. The strongest signals are observed in BRCA for RNA.
ACTBP13 data typeExpression analysisLineage consensusLineage of highest sampling consensus
RNABox plot8BRCA (6)view →
This table ranks reproducible tumor–normal expression differences for ACTBP13. A negative fold-change indicates higher expression in normal tissue than in tumor tissue. ACTBP13 shows higher tumor expression in BRCA, PAAD, COAD, UCEC, LIHC and STAD. The BRCA box plot shows higher ACTBP13 RNA expression in tumor versus normal tissue (log2 FC = +0.036, t-test p < 0.001).
LineageGenderStageFold-changepSampling consensus
BRCAAllAll+0.036<.0016view →
PAADFemaleAll+0.279.0254view →
COADMaleAll+0.063.0124view →
UCECAllAll+0.058.0174view →
LIHCAllAll+0.008.0043view →
STADAllII,III,IV+0.124.0232view →
Green = repressed in tumor. all 8 lineages →

ACTBP13-BRCA

Tumor-vs-normal expression box plot for ACTBP13 in BRCA.

Explore this plot interactively →

Cross-omics associations

This table shows molecular features associated with ACTBP13 in patient tissues and cancer cell lines. In patient samples, ACTBP13 shows the broadest associations at the RNA and protein expression levels, with ESCA recurring as the lineage with the largest associated feature set.
Associated data typeStrength (# associated data)Lineage of highest associated data
RNA
RNA9,174ESCA (3273)view →
Function (RNA)6,698KIRC (4660)view →